COMMON
NAME(S):
Black Walnut
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LATIN
NAME: Juglans nigra (Juglans cinerea)
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BRIEF
DESCRIPTION:
Juglans nigra and Juglans cinerea (Black Walnut and
Butternut) are traditionally known to have great medicinal properties, similar
to their European cousin (Juglans regia).
However, there are very few studies that illuminate the qualities of Juglans nigra specifically. Most of the scientific understanding
of Juglans nigra is inferred from
the study of Juglans cinerea/regia.
The
green hull of Black Walnut, along with the bark, has traditionally been used
to treat parasites, specifically worms.
Its action as an anti-parasitic drug can easily be remembered by the
natural history of the walnut family.
The roots of the walnut tree secrete a substance that is toxic to
other plant life (juglone). That
is why many plants cannot grow beneath a walnut tree. It keeps the ground
clear of other plants that might compete with it for growth, in the same way
that it kills parasites that compete for nutrients in the human body.
Walnut
hulls are also a “cathartic” medicine, known for their purging qualities, and
can been used to treat constipation.
It does this by acting on the liver, gallbladder, muscle fibers and
mucous membranes of the bowels. Lower
doses are thought to effectively tonify the mucous membranes of the bowels.
It
is thought that the anti-parasitic and bowel-tonifying action of walnut can
help promote skin health too (when skin lesions are related to constipation
or parasites).
The
nuts are a dietary source of healthy fatty acids. Traditionally recommended
that pregnant women consume the nut (NOT the leaves or the hulls), in order
to encourage the healthy development of the baby’s brain and other
organs. This is consistent with
what we know today about the positive relationship between healthy fatty
acids (omega 3 and 6 polyunsaturated fatty acids) and their role in the
cognitive development of children.
The
good-fat content of walnuts also makes them great food for the heart.
Walnuts
can help lower cholesterol and prevent plaques from forming in the
arteries. In 2003, The FDA
stated that eating
1.5 oz. per day of walnuts, as part of a diet low in saturated fat and
cholesterol, may reduce the risk of heart disease. Walnuts contain heart healthy nutrients such as
unsaturated fatty acids, fiber, potassium and folate. They are also high in vitamin E and
the potent antioxidant, ellagic acid.
Preliminary animal studies show that ellagic acid has potential
anti-cancer action.
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PARTS
USED: nut, hull,
husk, leaves, bark
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ACTIONS: anti-parasitic, purgative, cardio
protective (fats), nutritive
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INDICATIONS: parasites, eczema, constipation, pregnancy (nuts)
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BODY
SYSTEMS TREATED:
cardiovascular, skin, digestive
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PREPARATIONS: tincture, tea, powdered, whole food
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TRADITIONAL
USES: parasites,
lice, scabies, bowel tonification, food-source,
colour dye.
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SAFETY/CAUTIONS/INTERACTIONS: do not use while pregnant or
breast-feeding. Cross reactivity with other nut allergies.
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TRADITIONAL
REFERENCES:
2) Hutchens, Alma. Indian Herbology of North America.
3) Mitchell, William. Plant Medicine in Practice: Using
the Teachings of John Bastyr.
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MODERN
REFERENCES:
1) Feldman EB. The scientific evidence for a beneficial health
relationship between walnuts and coronary heart disease. J Nutr
2002;132:1062S-101S.
2) Zambon D, Sabate J, Munoz S, et al. Substituting walnuts for
monounsaturated fat improves the serum lipid profile of hypercholesterolemic
men and women. Ann Intern Med 2000;132:538-46.
3) Chisholm A, Mann J, Skeaff M, et al. A diet rich in walnuts
favourably influences plasma fatty acid profile in moderately hyperlipidaemic
subjects. Eur J Clin Nutr 1998;52:12-6.
4) Sabate J, Fraser GE, Burke K, et al. Effects of walnuts on
serum lipid levels and blood pressure in normal men. N Engl J Med
1993;328:603-7
5) J Nutr. 2001 Nov;131(11):2837-42.Walnut polyphenolics inhibit
in vitro human plasma and LDL oxidation.These results demonstrate that walnut
polyphenolics are effective inhibitors of in vitro plasma and LDL oxidation.
The polyphenolic content of walnuts should be considered when evaluating their
antiatherogenic potential.
6) Narayanan
BA, Geoffroy O, Willingham MC, Re GG, Nixon DW (March 1999). "p53/p21(WAF1/CIP1)
expression and its possible role in G1 arrest and apoptosis in ellagic acid
treated cancer cells". Cancer Lett. 136 (2):
215–21. doi:10.1016/S0304-3835(98)00323-1.
PMID 10355751.
7) Madal, Shivappurkar, Galati, and Stoner
(1988). "Inhibition of N-nitrosobenzymethylamine metabolism and DNA
binding in cultured rat esophagus by ellagic acid". Carcinogenesis
9 (7): 1313–1316. doi:10.1093/carcin/9.7.1313.
PMID 3383347.
8) Mandal and Stoner; Stoner, GD (1990).
"Inhibition of N-nitrosobenzymethylamine-induced esophageal
tumorigenesis in rats by ellagic acid". Carcinogenesis 11
(1): 55–61. doi:10.1093/carcin/11.1.55. PMID 2295128.
9)
www.naturaldatabase.therapeuticresearch.com
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